Cerebral Infarction
26 drugs1,040 FAERS reportsMedDRA PT
GET
/api/v1/reaction/cerebral-infarctionReference statistics only. FAERS reports are voluntarily submitted and are not incidence rates, safety signals, or causal evidence. The drugs below are ranked by how often this reaction appears on FAERS reports for each drug — high share does not mean the drug caused the reaction, and absence from this list does not mean the reaction never occurred. This is not a symptom checker.
DefinitionNLM MeSH D002544
The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
Source: U.S. National Library of Medicine — MeSH (Cerebral Infarction)
Drugs reporting Cerebral Infarction (26)
Share = reports listing this reaction ÷ total matched reports for the drug. — means the upstream totals query failed at ingest and the denominator is unknown.
- 1Acetone125%
- 2Benzopyrene125%
- 3Carbon Tetrachloride125%
- 4Phenolphthalein125%
- 5Trichloroethylene125%
- 6Phenylacetate111%
- 7Andexanet Alfa547.7%
- 8Glutamate17.7%
- 9Trypan Blue36.5%
- 10Edaravone956.4%
- 11Sulconazole14.5%
- 12Edoxaban2524.0%
- 13Argatroban503.4%
- 14Daprodustat573.2%
- 15Butamben13.1%
- 16Vadadustat72.8%
- 17Cobaltous Chloride12.7%
- 18Exametazime12.6%
- 19Anagrelide512.3%
- 20Flucytosine292.2%
- 21Lecanemab702.1%
- 22Idarucizumab292.0%
- 23Alogliptin721.9%
- 24Tenecteplase271.8%
- 25Ethiodized Oil211.7%
- 26Alteplase2121.4%
Related reactions
Other reactions most often co-reported on the same drug set. Ranked by Jaccard similarity over drug-id sets — pure data-derived co-occurrence, not a clinical relationship.
- Cerebral Haemorrhage8 shared · 0.21
- Stupor5 shared · 0.19
- Apnoea5 shared · 0.15
- Ischaemic Stroke4 shared · 0.13
- Haemorrhage Intracranial4 shared · 0.13
- Haemorrhagic Transformation Stroke3 shared · 0.12
- Influenza Like Illness6 shared · 0.11
- Pneumonia Aspiration3 shared · 0.09
- Gastrointestinal Haemorrhage5 shared · 0.08
- Coagulopathy3 shared · 0.08
Literature
Recent PubMed references pinned to Cerebral Infarction as a MeSH major topic. About the reaction term itself — not about any specific drug.
- Nuclear Factor Erythroid 2-Related Factor 2-Dependent Ferroptosis Suppression by Salvianolic Acid B Preserves Microvascular Integrity and Reduces Risk Factors for Hemorrhagic Transformation After Cerebral Infarction.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2026 · Liang J, Zhao X, Xiong R, et al.PMID 42186809DOI 10.1096/fj.202504432R
- [A case of acute ethylene glycol poisoning complicated with acute cerebral infarction].Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational diseases · 2026 · Chen JY, Guo H, Yan K, et al.PMID 42185104DOI 10.3760/cma.j.cn121094-20240918-00431
- Prognostic Analysis of Butylphthalide Therapy Guided by CYP2C19 Genotype in Patients with Acute Cerebral Infarction.Journal of visualized experiments : JoVE · 2026 · Ye L, Ruan Q, Zhang J, et al.PMID 42184267DOI 10.3791/69165
- White Matter Microstructural Injury in Patients With Acute Single Subcortical Infarction: A Preliminary Tract-Based Spatial Statistics With Atlas-Based Analysis.Neural plasticity · 2026 · Zhu Z, Shen T, Xiong J, et al.PMID 42178813DOI 10.1155/np/1200067
- NOTCH3 signal activation by its extracellular domain accumulation in an iPSC line newly established from a CADASIL patient.Human cell · 2026 · Sun R, Zhao Z, Che N, et al.PMID 42177738DOI 10.1007/s13577-026-01391-3
- Risk factors for poor prognosis in patients with cortical laminar necrosis and establishment of their prediction model: A retrospective cohort study.Medicine · 2026 · Wang W, Zhang X, Zhang M, et al.PMID 42175459DOI 10.1097/MD.0000000000048871
Note. FAERS reports are voluntarily submitted and are NOT incidence rates, signals, or causal evidence. Counts reflect reporting volume, not how often a reaction occurs. Reference statistics only.